Miranda Lambert is having a moment

Miranda Lambert and her trainer do circuit training, lunges, and crunches and walk every morning for 30 minutes.

STORY HIGHLIGHTS

• Lambert eats a lot of protein to stay energized on the road
• Lambert and fiance Blake Shelton have seven dogs, no time for kids
• "The hard part is I'm a normal-size girl, and I'm fine with that," Lambert says

(Health.com) -- Miranda Lambert may have forgotten to thank her fianc�, fellow country music star Blake Shelton, when she won her first Grammy this February.

But when we caught up with the 27-year-old star on her tour, Blake was top of mind. In fact, the Texas native was thrilled to chat about everything, from her energy secrets to how she's getting into peak shape to swap "I do's" with Blake this month.

Q: You're in Evansville, Indiana, now. Do you ever get confused about which city you're in?

A: Yes! They usually write where we are on the set list, because I've made the mistake of saying the wrong state before. I was like, "Hello, Ohio!" And they just kind of looked at me like my head was on fire. [Laughs.]

Q: And how do you keep your energy up on stage?

A: I eat a lot of protein and eat pretty clean. Junk food drags you down. I try not to have anything less than two hours before I go on stage, because I don't want to be up there all bloated. I basically have a grilled-chicken salad after the show, and that fills me up.

Health.com: 9 low-cal chicken salad recipes

Q: You have a trainer with you on the road, right?

A: Yeah, I wanted to get in better shape and wanted to keep it going for the wedding. We do a lot of circuit training, lunges, and crunches. And every morning we walk for 30 minutes.

Q: Your parents were private eyes who ran an agency from home. It sounds like a TV show!

A: It does. But to us [back then], it was just my parents' job. I did surveillance a lot, which sounds exciting, but it never was. If there was no babysitter, I'd have to go, and my brother and I would just lie in the backseat and color for 15 hours while my mom watched the house.

Q: Did you know what your parents were doing, exactly?

A: In a way, I had such a normal life. But in another way, I saw things most kids don't see. Our dinner conversations weren't about school and homework; they were about child custody cases or divorce. When I was a teenager, my parents took in victims of domestic abuse, which was definitely eye-opening. I had to share my room with a woman and her kids who had been abused.

Health.com: 8 reasons to make time for family dinner

Q: What was the healthiest thing about your childhood?

A: We had a farm and a garden, and for about two years we literally lived off the land. We raised rabbits and chickens, and also had our own hogs that we slaughtered. We didn't really go to the store for anything besides milk. My mom would say, "Go pick out what you want for dinner," and we'd go in the garden and get whatever vegetables we wanted. It was really old-school and really awesome.

Q: Was it hard slaughtering animals that were your pets?

A: Dad would give us two rabbits as our pets, or we'd have one pig we could name. They explained that not every animal was a pet -- some were providing for our family. It sounds weird to other people, but I look at it like, there are some animals you feed and some animals that feed you.

Q: Are you a good cook?

A: [Blake and I are] both pretty good. We have a lot of catfish, and everybody knows the best way to eat catfish is fried. I tried to be healthy and bake it once, which was horrible. We ordered a pizza!

Health.com: 13 easy pizza recipes

Q: How did you know that Blake was the one?

A: The first day I met him, I knew he was really special. We balance each other out. He's constantly joking, and I'm more serious. He can make me laugh at any point. Especially when I'm trying to be mad, he'll go, "Are we in a fight?" And then I'll start laughing and go, "I guess not now."

Q: He must understand the pressure of being in the public eye. It's not enough anymore to just be a singer -- you have to look great, too. Is that tough?

A: Absolutely. The bigger you get, the more pressure you have to deal with. The hard part is I'm a normal-size girl, and I'm fine with that. All these stars are so tiny, and they spend their lives trying to be skinny. I want to look my best, but I'm not a model. I'm not an actress. I'm representing normal girls. It's OK to have a little bit of curve. I'm happy with my body. So many girls come up to me and say, "Thank you for being normal," and I'm proud of that.

Health.com: Find your feel great weight

Q: Do you guys want kids?

A: We have seven dogs, so we're just taking care of them. They're all rescues, and all crazy-looking. Every animal is named after a song or an artist -- Delta, Delilah, Cher, Jessi, Waylon, Virginia Bluebell, and Black Betty. We have so much going on right now, we're gonna wait awhile. The dogs are really good birth control!

Copyright Health Magazine 2010

Source: http://rss.cnn.com/~r/rss/cnn_health/~3/xH5Plzyzm2I/index.html

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Omega-3s Linked To Higher Risk Of Aggressive Prostate Cancer

Featured Article
Academic Journal
Main Category: Prostate / Prostate Cancer
Also Included In: Nutrition / Diet;��Heart Disease;��Men's health
Article Date: 26 Apr 2011 - 8:00 PDT email to a friend � printer friendly � opinions �

Current Article Ratings:

Patient / Public:		4.2 (5 votes)
Healthcare Prof:		2.6 (5 votes)
Article Opinions:	�4 posts

US researchers were stunned to discover that higher blood levels of omega-3 fatty acids, normally promoted as good for the heart, were linked to higher risk of aggressive prostate cancer, and conversely, raised levels of trans-fats, considered bad for the heart, were linked to a lower risk.

However, neither of the fats was found to be linked to risk of low-grade prostate cancer.

These were the findings of the largest study ever to look at links between dietary fats and prostate cancer risk.

Lead and corresponding author Theodore M. Brasky, of the Fred Hutchinson Cancer Research Center in Seattle, Washington, told the press:

"We were stunned to see these results and we spent a lot of time making sure the analyses were correct."

He and colleagues in the Hutchinson Center's Public Health Sciences Division, and also from the University of Texas Health Science Center at San Antonio and the National Cancer Institute, analyzed a subset of data on more than 3,4000 men taking part in the Prostate Cancer Prevention Trial, a large nationwide randomized clinical trial, involving more than 19,000 men aged 55 and over, that tested the performance of the drug finasteride in preventing prostate cancer.

Their paper was published this week in the American Journal of Epidemiology. Brasky, a postdoctoral research fellow in the Hutchinson Center's Cancer Prevention Program, said:

"Our findings turn what we know - or rather what we think we know - about diet, inflammation and the development of prostate cancer on its head and shine a light on the complexity of studying the association between nutrition and the risk of various chronic diseases."

Brasky and colleagues found that men with the highest blood levels of docosahexaenoic acid, or DHA, an omega-3 fatty acid that lowers inflammation and is common in fatty fish, had 2.5 times the risk of developing aggressive, high-grade prostate cancer compared to men with the lowest blood leves of DHA.

They also found that men with the highest blood percentages of trans-fatty acids, which have been linked to inflammation and heart disease, and are common in processed foods made with partially hydrogenated vegetable oils, had a 50% reduced risk of developing high-grade prostate cancer.

In contrast, they found neither of the two types of fatty acids was linked to raised risk of low-grade prostate cancer.

They also found that omega-6 fatty acid, common in most vegetable oils, and which has been linked to inflammation and heart disease, was not linked to raised risk of either high-grade or low-grade prostate cancer.

The results stunned the researchers, whose prime motive was to test the idea that concentrations of these various types of fatty acids in the blood would show some relationship with prostate cancer risk, in a similar pattern to their various links with inflammation and heart disease risk.

"Specifically, we thought that omega-3 fatty acids would reduce and omega-6 and trans-fatty acids would increase prostate cancer risk," said Brasky.

Why omega-3s should be linked to raised risk of high-grade prostate cancer is a mystery to the researchers.

Brasky said they need to do more research on it, but speculated that perhaps omega-3s affect biological mechanisms other than inflammation, and these have a bigger effect on the development of some prostate cancers.

Although many nutritionists and doctors recommend 450 mg daily dose of omega-3 DHA as part of a healthy diet, there are currently no official guidelines in the US on recommended daily allowance.

Half of the participants in the nationwide trial developed prostate cancer during the course of the study, and half did not. The trial is considered unique because all participants underwent biopsy to confirm the presence or absence of prostate cancer.

Few of the participants took fish oil supplements, the most common non-dietary source of omega-3 fatty acids, which are known to prevent heart disease and other diseases caused by inflammation. Most of the participants got their omega-3s from consuming fish.

Brasky and colleagues don't think that these findings should discourage men from continuing to get their omega-3s from oily fish or food supplements.

"Overall, the beneficial effects of eating fish to prevent heart disease outweigh any harm related to prostate cancer risk," said Brasky, stressing that the study highlights the complexity of nutrition and its links with disease risk, and we need to study them much more rigorously before jumping to conclusions.

Funds from the National Cancer Institute helped pay for the study.

"Serum Phospholipid Fatty Acids and Prostate Cancer Risk: Results From the Prostate Cancer Prevention Trial."
Theodore M. Brasky, Cathee Till, Emily White, Marian L. Neuhouser, Xiaoling Song, Phyllis Goodman, Ian M. Thompson, Irena B. King, Demetrius Albanes, and Alan R. Kristal
Am. J. Epidemiol. first published online 24 April 2011
DOI:10.1093/aje/kwr027

Additional source: Fred Hutchinson Cancer Research Center.

Written by: Catharine Paddock, PhD
Copyright: Medical News Today
Not to be reproduced without permission of Medical News Today

Please rate this article: (Hover over the stars then click to rate)

Patient / Public:

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Omega 3s And Prostate Cancer - Told You So

posted by keira on 26 Apr 2011 at 9:37 am

told u those baby foods fortified with tuna oil were a bad idea!!!

| post followup | alert a moderator |

Wish they studied non-animal sources of Omegas

posted by Greg on 26 Apr 2011 at 9:51 am

There is another factor here: Fish protein. I wish this study included omega-3s extracted from flax or hemp seed. Some other studies indicate animal protein may cause cancer.

| post followup | alert a moderator |

Omega 3

posted by Dr. Jennifer Adolph on 26 Apr 2011 at 6:06 pm

The description of the study does not specify dietary intake of Omega-3 fatty acids, only blood levels. Therefore it is impossible to evaluate whether the DHA levels are 1) causative or 2) raised as a sequela; of agressive prostate cancer.

| post followup | alert a moderator |

PCBs and other contaminants?

posted by PW on 26 Apr 2011 at 7:59 pm

I recall reading something that indicated that PCBs and other dioxin-like contaminants cause not only prostate cancer at younger ages, but more often in aggressive forms. There is a third variable problem and the correlation found between omega 3s and cancer does not prove causation. That is, it may be PCBs (or some other unidentified variable) in fish contribute to the cancer rates, not the omega 3 fatty acids.

| post followup | alert a moderator |

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Source: http://www.medicalnewstoday.com/articles/223373.php

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What We're Reading This Week

health:�blog�

NEWS APRIL 22

Kristen Dold

More Peas, Please
It's the freshest recipe you've ever seen. And the one most fit for spring. And you get to smash something! Curious, yet? Peas and fava beans with fresh mozz on cibabtta! Grind it, spread it, eat it. YUM.
Via The Cilantropist

The Perfect Picnic
There are regular picnics. And then there are everything-matches-so-beautifully picnics with fresh chickpea and tabouleh salad, individual size spinach tortilla quiche, and pineapple upside down cake balls (those exist!). Not the same. Not even close. Via Hostess with the Mostess

Earth Day Dessert
Try making your panna cotta with agar agar instead of gelatin--it's made from red algae instead of animal collagen (weird yes, but sustainable!). You'll never taste the difference. Try this vanilla coffee Kahlua panna cotta with peanut butter dots! Via Lisa is Cooking

Sex Buzz
Next time you hit your local pharmacy for a gallon of milk and some gum, why not grab a vibrator? The battery-powered pleasure devices, which used to be confined to small sex shops and online sites, now line the shelves at Walgreens and other mainstream drugstores. Via NY Times

CPR U Ready?
People are buzzing about the 9-year-old boy who helped save his little sister?s life by performing CPR on her after she fell into a pool. (He said he picked it up from watching Blackhawk Down!) Would you know what to do in an emergency? Get a speedy tutorial with this quick YouTube video. Via LA Times

What?s Lurking In Your Meat?
You all know that uncooked meat can carry pathogens like salmonella, listeria, and E. coli, but a new study says that almost half of the meat you buy at grocery stores is contaminated with Staph (Staphylococcus aureus) bacteria! Click here for a refresher on food safety measures.
Via Reuters

Tags: headlines

Last Updated: April 22, 2011 - 12:24pm

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Source: http://www.womenshealthmag.com/health/news-april-22

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lisinopril, Zestril, Prinivil

Pharmacy Author: Omudhome Ogbru, PharmD
Medical and Pharmacy Editor: Jay W. Marks, MD

GENERIC NAME: lisinopril

BRAND NAME: Zestril, Prinivil

DRUG CLASS AND MECHANISM: Lisinopril is an angiotensin converting enzyme (ACE) inhibitor used for treating high blood pressure, heart failure and for preventing kidney failure due to high blood pressure and diabetes. Other ACE inhibitors include enalapril (Vasotec), quinapril (Accupril), captopril (Capoten), fosinopril (Monopril), benazepril (Lotensin), ramipril (Altace), moexipril (Univasc) and trandolapril (Mavik).

ACE is important because it is an enzyme responsible for producing the chemical, angiotensin II. Angiotensin II causes muscles in most arteries, including the arteries of the heart, to contract, thereby narrowing the arteries and elevating blood pressure. ACE inhibitors such as lisinopril lower blood pressure by reducing the production of angiotensin II, thereby relaxing arterial muscle and enlarging arteries. When the blood pressure is lower, the heart - including the failing heart - does not have to work as hard to pump blood. The arteries supplying the heart with blood also enlarge during treatment with ACE inhibitors. This increases the flow of blood and oxygen to the heart, further improving the ability of the heart to pump blood.

The effects of ACE inhibitors are particularly beneficial to people with congestive heart failure. In the kidneys, the narrowing of the arteries by angiotensin II decreases blood flow and damages the kidneys. ACE inhibitors enlarge and reduce the blood pressure in the arteries going to the kidney. This reduces damage to the kidneys caused by the high blood pressure. The FDA approved lisinopril in December 1987.

PRESCRIPTION: Yes

GENERIC AVAILABLE: Yes

PREPARATIONS: Tablet: 2.5, 5, 10, 20, 30 and 40 mg

STORAGE: Lisinopril should be stored in a dry place at 15-30 C (59-86 F).

PRESCRIBED FOR: Lisinopril is used to treat elevated blood pressure, heart failure and to improve survival after a heart attack (myocardial infarction). Like other ACE inhibitors it is also used for preventing kidney failure due to high blood pressure or diabetes even though it is not FDA approved for this use.

DOSING: The starting dose of lisinopril is 5 mg daily, and the effective dose range for treating heart failure is 5-20 mg daily. The dose can be increased by 10 mg every 2 weeks to achieve the maximum effect.

The starting dose of lisinopril for treating high blood pressure is 10 mg daily. The usual dose range is 20-40 mg daily. A dose of 80 mg is not much more effective than 40 mg.

Treatment of heart attack (myocardial infarction) is started with individual doses of 5 mg followed by 5 mg after 24 hours, 10 mg after 48 hours and then 10 mg daily. Treatment is continued for 6 weeks.

DRUG INTERACTIONS: In general, lisinopril should not be taken with potassium supplements or diuretics that conserve potassium, for example, hydrochlorothiazide/triamterene (Dyazide), since blood potassium levels may rise to dangerous levels.

Report Problems to the Food and Drug Administration � You are encouraged to report negative side effects of prescription drugs to the FDA. Visit the FDA MedWatch website or call 1-800-FDA-1088.

Source: http://www.medicinenet.com/guide.asp?s=rss&a=862&k=Womens_Health_General

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Health Tip: Ease Baby's Stuffy Nose

[unable to retrieve full-text content]Title: Health Tip: Ease Baby's Stuffy Nose
Category: Health News
Created: 4/28/2011 8:05:00 AM
Last Editorial Review: 4/28/2011

Source: http://www.medicinenet.com/guide.asp?s=rss&a=143675&k=Womens_Health_General

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Researchers link spring births to anorexia

?People born in spring are 'more likely' to become anorexic,? reported The Independent. The newspaper said the finding comes from the first large-scale study of the link between anorexia and the season of birth.

This study pooled data on birth dates from four UK studies of around 1,300 people with anorexia nervosa and compared these to the distribution of births in the general population. The researchers found that more people who were born between March and June went on to develop anorexia than could be expected when compared to the patterns of birth seen in the general population.

The researchers propose several theories to explain this association, including the mother?s diet during pregnancy, seasonal infections such as flu, and climate, including temperature, rainfall and sunshine levels.

This research highlights increasing interest in how the environment of the developing baby may influence their likelihood of being affected by certain diseases later in life. The effect observed here was small however, and more research and further analysis will be needed to establish just how robust this association is and to investigate the possible causes behind it. The media also includes comments from experts which give these results some context. The experts say that anorexia is a very complex disorder and that several factors contribute to its development.

�

Where did the story come from?

The study was carried out by researchers from the Wellcome Trust Centre for Human Genetics at the University of Oxford. The work was supported by the Wellcome Trust. The study was published as a short report in the peer-reviewed journal The British Journal of Psychiatry.

Both The Independent and BBC News report the study accurately. The Independent gives several theories explaining the difference in rates of anorexia between people born in different seasons. However, the study was only set up to find associations and it remains unclear why there might be a difference between seasonal rates. The BBC appropriately highlights that other academics have said that the effect was small and the disorder has many causes.

�

What kind of research was this?

This was a meta-analysis of data from several cohort studies aimed at investigating whether the season in which someone is born affects their risk of developing anorexia nervosa. The researchers combined and compared data from four separate studies. They looked at the distribution of birth days of people who developed anorexia and compared them with those of the general UK population.

The methods that the researchers used are only briefly described in the research paper. The paper does not describe in detail what is already known about this topic, which studies were excluded, or give the separate results of the one Scottish and three English studies that were included in the analysis. As three of these four studies separately showed non-significant results, larger sample sizes will be required in any future research.

�

What did the research involve?

To find the articles for their analysis, the researchers searched a scientific literature database called PubMed. They say that they included studies from the UK only, because country-specific factors such as different social and nutritional habits, rates of disease, birth trends and latitude might have confounded the results.

They found four relevant studies for inclusion in their meta-analysis. The largest was published in 2001 and was conducted in Scotland, recruiting 446 people and then following them between 1965 and 1997. This study found that more people who were born between April and June went on to develop anorexia than usual, and that cases peaked in the spring months, while sinking in the autumn months.

The other three studies were published between 2002 and 2007 and were all conducted in England, ranging in size from 195 to 393 people. Although these three studies showed a similarly high number of April?June births, the difference between the number of babies who later developed anorexia who were born in the ?peak? spring months and the ?trough? autumn months was not significant.

The researchers pooled the results using standard and non-standard statistical techniques. They compared the anorexia nervosa birth rates in the first half of the year with the second half. They also compared the rates of anorexia births in spring (March to June) and autumn (September to October) with those of the general population born between 1950 and 1980. The general population rates were obtained from the UK Office for National Statistics and comprised almost 22 million births over a similar period (1950-1980).

�

What were the basic results?

The researchers say they found a 15% excess of anorexia births from March to June (odds ratio [OR] 1.15, 95% Confidence Interval [CI] 1.03 to 1.29). This means that, for example, if the background rate of anorexia is 20 per 4,000 births per month then one could expect 23 (15% more) in the months March to June.

In contrast, from September to October there was a 20% deficit from (OR 0.8, 95% CI 0.68 to 0.94).

The distribution of births in the anorexia group was significantly different from that of the general population. Analysis showed that rates of people born with anorexia were higher in the first half of the year compared with the second (OR� 1.13, 95% CI 1.01 to 1.26).

�

How did the researchers interpret the results?

The researchers say that their results indicate that environmental risk factors during pregnancy or immediately after birth affect whether someone goes on to develop anorexia in later life. They say that further identification of these risk factors will be ?important for disease prevention strategies?.

�

Conclusion

This research highlights increasing interest in how the environment of the developing baby might influence their likelihood of being affected by certain diseases later in life. The researchers refer to this as the ?fetal origin of adult disease? hypothesis. This study appears to support this theory, but further study is needed to confirm this association and investigate the mechanism behind it.

The researchers mention several environmental factors that vary throughout the year that they considered most likely to be linked to the development of anorexia:

• seasonal changes in temperature
• sunlight exposure and consequent vitamin D levels
• maternal nutrition (which may differ during winter months)
• levels of common infections, such as colds

The mention of vitamin D levels is also picked up by the newspapers as this has been linked to other psychiatric diseases, including schizophrenia, and neurological conditions, such as multiple sclerosis. However, these researchers appear to suggest that low vitamin D levels may be a result of psychiatric illness rather than a cause.

Overall, this study indicates that the seasons may have a small effect on rates of anorexia in babies born during the spring months. The results do not indicate what the absolute risk of developing the condition might be for a person born in the spring. More research and further analysis of research in other countries will be needed to establish just how robust this association is and to investigate the possible causes behind it.

Links To The Headlines

Anorexia nervosa 'link to spring birth?.�BBC News, April 28 2011

People born in spring are 'more likely' to become anorexic.�The Independent, April 28 2011

�

Links To Science

Disanto G, Handel AE, Para AE, et al.�Season of birth and anorexia nervosa. The British Journal of Psychiatry 2011, Published ahead of print

Source: http://www.nhs.uk/news/2010/04April/Pages/spring-births-and-anorexia.aspx

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Omega-3s Linked To Higher Risk Of Aggressive Prostate Cancer

Featured Article
Academic Journal
Main Category: Prostate / Prostate Cancer
Also Included In: Nutrition / Diet;��Heart Disease;��Men's health
Article Date: 26 Apr 2011 - 8:00 PDT email to a friend � printer friendly � opinions �

Current Article Ratings:

Patient / Public:		4 (4 votes)
Healthcare Prof:		2.6 (5 votes)
Article Opinions:	�4 posts

US researchers were stunned to discover that higher blood levels of omega-3 fatty acids, normally promoted as good for the heart, were linked to higher risk of aggressive prostate cancer, and conversely, raised levels of trans-fats, considered bad for the heart, were linked to a lower risk.

However, neither of the fats was found to be linked to risk of low-grade prostate cancer.

These were the findings of the largest study ever to look at links between dietary fats and prostate cancer risk.

Lead and corresponding author Theodore M. Brasky, of the Fred Hutchinson Cancer Research Center in Seattle, Washington, told the press:

"We were stunned to see these results and we spent a lot of time making sure the analyses were correct."

He and colleagues in the Hutchinson Center's Public Health Sciences Division, and also from the University of Texas Health Science Center at San Antonio and the National Cancer Institute, analyzed a subset of data on more than 3,4000 men taking part in the Prostate Cancer Prevention Trial, a large nationwide randomized clinical trial, involving more than 19,000 men aged 55 and over, that tested the performance of the drug finasteride in preventing prostate cancer.

Their paper was published this week in the American Journal of Epidemiology. Brasky, a postdoctoral research fellow in the Hutchinson Center's Cancer Prevention Program, said:

"Our findings turn what we know - or rather what we think we know - about diet, inflammation and the development of prostate cancer on its head and shine a light on the complexity of studying the association between nutrition and the risk of various chronic diseases."

Brasky and colleagues found that men with the highest blood levels of docosahexaenoic acid, or DHA, an omega-3 fatty acid that lowers inflammation and is common in fatty fish, had 2.5 times the risk of developing aggressive, high-grade prostate cancer compared to men with the lowest blood leves of DHA.

They also found that men with the highest blood percentages of trans-fatty acids, which have been linked to inflammation and heart disease, and are common in processed foods made with partially hydrogenated vegetable oils, had a 50% reduced risk of developing high-grade prostate cancer.

In contrast, they found neither of the two types of fatty acids was linked to raised risk of low-grade prostate cancer.

They also found that omega-6 fatty acid, common in most vegetable oils, and which has been linked to inflammation and heart disease, was not linked to raised risk of either high-grade or low-grade prostate cancer.

The results stunned the researchers, whose prime motive was to test the idea that concentrations of these various types of fatty acids in the blood would show some relationship with prostate cancer risk, in a similar pattern to their various links with inflammation and heart disease risk.

"Specifically, we thought that omega-3 fatty acids would reduce and omega-6 and trans-fatty acids would increase prostate cancer risk," said Brasky.

Why omega-3s should be linked to raised risk of high-grade prostate cancer is a mystery to the researchers.

Brasky said they need to do more research on it, but speculated that perhaps omega-3s affect biological mechanisms other than inflammation, and these have a bigger effect on the development of some prostate cancers.

Although many nutritionists and doctors recommend 450 mg daily dose of omega-3 DHA as part of a healthy diet, there are currently no official guidelines in the US on recommended daily allowance.

Half of the participants in the nationwide trial developed prostate cancer during the course of the study, and half did not. The trial is considered unique because all participants underwent biopsy to confirm the presence or absence of prostate cancer.

Few of the participants took fish oil supplements, the most common non-dietary source of omega-3 fatty acids, which are known to prevent heart disease and other diseases caused by inflammation. Most of the participants got their omega-3s from consuming fish.

Brasky and colleagues don't think that these findings should discourage men from continuing to get their omega-3s from oily fish or food supplements.

"Overall, the beneficial effects of eating fish to prevent heart disease outweigh any harm related to prostate cancer risk," said Brasky, stressing that the study highlights the complexity of nutrition and its links with disease risk, and we need to study them much more rigorously before jumping to conclusions.

Funds from the National Cancer Institute helped pay for the study.

"Serum Phospholipid Fatty Acids and Prostate Cancer Risk: Results From the Prostate Cancer Prevention Trial."
Theodore M. Brasky, Cathee Till, Emily White, Marian L. Neuhouser, Xiaoling Song, Phyllis Goodman, Ian M. Thompson, Irena B. King, Demetrius Albanes, and Alan R. Kristal
Am. J. Epidemiol. first published online 24 April 2011
DOI:10.1093/aje/kwr027

Additional source: Fred Hutchinson Cancer Research Center.

Written by: Catharine Paddock, PhD
Copyright: Medical News Today
Not to be reproduced without permission of Medical News Today

Please rate this article: (Hover over the stars then click to rate)

Patient / Public:

or

Health Professional:

---

Note: Any medical information published on this website is not intended as a substitute for informed medical advice and you should not take any action before consulting with a health care professional. For more information, please read our terms and conditions.

---

Omega 3s And Prostate Cancer - Told You So

posted by keira on 26 Apr 2011 at 9:37 am

told u those baby foods fortified with tuna oil were a bad idea!!!

| post followup | alert a moderator |

Wish they studied non-animal sources of Omegas

posted by Greg on 26 Apr 2011 at 9:51 am

There is another factor here: Fish protein. I wish this study included omega-3s extracted from flax or hemp seed. Some other studies indicate animal protein may cause cancer.

| post followup | alert a moderator |

Omega 3

posted by Dr. Jennifer Adolph on 26 Apr 2011 at 6:06 pm

The description of the study does not specify dietary intake of Omega-3 fatty acids, only blood levels. Therefore it is impossible to evaluate whether the DHA levels are 1) causative or 2) raised as a sequela; of agressive prostate cancer.

| post followup | alert a moderator |

PCBs and other contaminants?

posted by PW on 26 Apr 2011 at 7:59 pm

I recall reading something that indicated that PCBs and other dioxin-like contaminants cause not only prostate cancer at younger ages, but more often in aggressive forms. There is a third variable problem and the correlation found between omega 3s and cancer does not prove causation. That is, it may be PCBs (or some other unidentified variable) in fish contribute to the cancer rates, not the omega 3 fatty acids.

| post followup | alert a moderator |

---

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If you write about specific medications or operations, please do not name health care professionals by name.

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